[Hepatitis A and hepatitis B. Clinical, epidemiological and differential immunological elements]. 1977

L Ortona, and E Pizzigallo, and G Federico, and G Camilli, and V Laghi

The Authors have already related in various papers the results of their research on humoral and cellular immunity in the course of viral hepatitis in relation to the behaviour of HBsAg determined with various methods and recently with the radio-immune assay. Since the high sensitivity of this latter technique consents a sufficiently precise differentiation of hepatitis into HBAg-positive (hepatitis B) and HBAg-negative (hepatitis A), the Authors have intended studying in this work the eventual clinical epidemiological and immunological differences between the two types of hepatitis, analyzing the results of their previous studies, extending the case data and gathering numerous other personal observations, clinical and laboratory, not yet published. The study was limited to 136 patients with viral hepatitis in wwhich the test for HBsAg was effected with RIA: of these 58 were HBAg+ and 78 HBAg-. Initially is discussed the incidence of two types of hepatitis in relation to age and sex and then the comparative incidence between them of the various routes of contagion. Particularly emphasized is the possibility of direct transmission of HBAg+ hepatitis, often responsible for infection within the family. The comparative determination of transaminases, bilirubin, immunoglobulins and complement has revealed some interesting variations in the behaviour of the two types of hepatitis. Instead, cellular immunity, studied by the rosette E technique, appeared depressed in the initial phase of sickness in both types of hepatitis. Space is dedicated to the behaviour of HBsAg in acute HBAg+ hepatitis in relation to the immunoglobulins and complement and in relation to the normalization of the transaminases and bilirubinemia. After a few brief observations on the frequent appearance of a joint symptomatology in the course of viral hepatitis, particularly in the HBAg+ form, the Authors report the data relative the evolution of the hepatitis cases examined. The percentage of a complete cure are about equal between hepatitis A and hepatitis B even recovery is significantly faster in the first type of hepatitis (HBAg-).

UI MeSH Term Description Entries
D007111 Immunity, Cellular Manifestations of the immune response which are mediated by antigen-sensitized T-lymphocytes via lymphokines or direct cytotoxicity. This takes place in the absence of circulating antibody or where antibody plays a subordinate role. Cell-Mediated Immunity,Cellular Immune Response,Cell Mediated Immunity,Cell-Mediated Immunities,Cellular Immune Responses,Cellular Immunities,Cellular Immunity,Immune Response, Cellular,Immune Responses, Cellular,Immunities, Cell-Mediated,Immunities, Cellular,Immunity, Cell-Mediated,Response, Cellular Immune
D007136 Immunoglobulins Multi-subunit proteins which function in IMMUNITY. They are produced by B LYMPHOCYTES from the IMMUNOGLOBULIN GENES. They are comprised of two heavy (IMMUNOGLOBULIN HEAVY CHAINS) and two light chains (IMMUNOGLOBULIN LIGHT CHAINS) with additional ancillary polypeptide chains depending on their isoforms. The variety of isoforms include monomeric or polymeric forms, and transmembrane forms (B-CELL ANTIGEN RECEPTORS) or secreted forms (ANTIBODIES). They are divided by the amino acid sequence of their heavy chains into five classes (IMMUNOGLOBULIN A; IMMUNOGLOBULIN D; IMMUNOGLOBULIN E; IMMUNOGLOBULIN G; IMMUNOGLOBULIN M) and various subclasses. Globulins, Immune,Immune Globulin,Immune Globulins,Immunoglobulin,Globulin, Immune
D007558 Italy A country in southern Europe, a peninsula extending into the central Mediterranean Sea, northeast of Tunisia. The capital is Rome. Sardinia
D008297 Male Males
D008875 Middle Aged An adult aged 45 - 64 years. Middle Age
D011863 Radioimmunoassay Classic quantitative assay for detection of antigen-antibody reactions using a radioactively labeled substance (radioligand) either directly or indirectly to measure the binding of the unlabeled substance to a specific antibody or other receptor system. Non-immunogenic substances (e.g., haptens) can be measured if coupled to larger carrier proteins (e.g., bovine gamma-globulin or human serum albumin) capable of inducing antibody formation. Radioimmunoassays
D005260 Female Females
D006506 Hepatitis A INFLAMMATION of the LIVER in humans caused by a member of the HEPATOVIRUS genus, HUMAN HEPATITIS A VIRUS. It can be transmitted through fecal contamination of food or water. Hepatitis, Infectious,Infectious Hepatitis,Hepatitides, Infectious,Infectious Hepatitides
D006509 Hepatitis B INFLAMMATION of the LIVER in humans caused by a member of the ORTHOHEPADNAVIRUS genus, HEPATITIS B VIRUS. It is primarily transmitted by parenteral exposure, such as transfusion of contaminated blood or blood products, but can also be transmitted via sexual or intimate personal contact. Hepatitis B Virus Infection
D006511 Hepatitis B Antigens Antigens of the virion of the HEPATITIS B VIRUS or the Dane particle, its surface (HEPATITIS B SURFACE ANTIGENS), core (HEPATITIS B CORE ANTIGENS), and other associated antigens, including the HEPATITIS B E ANTIGENS. HBAg,Hepatitis B Antigen,Antigen, Hepatitis B,Antigens, Hepatitis B,B Antigen, Hepatitis,B Antigens, Hepatitis

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