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We report an impressive decline in plasma lipid resistance to oxidation during Triton-WR-1339-induced hyperlipidemia in rats. This decline is associated with a modification in the balances between alpha-tocopherol and lipids and alpha-tocopherol and ascorbate. These results are consistent with a weak resistance of accumulated native lipoproteins in plasma to oxidation, during a 6-hour time course, and they suggest a misunderstood role of lipoprotein catabolic enzymes: to improve this characteristic. Conclusively, the results lead us to propound Triton-induced hyperlipidemia as an original model for studying the balance impairment between antioxidants and oxidizable substrates.
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L G Alcindor
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Pharmaceutical research,
H Antébi, and
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L G Alcindor
March 1997,
The American journal of physiology,
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March 1996,
Lipids,
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June 2003,
Free radical research,
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October 2005,
Biochemical pharmacology,
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Bulletin of experimental biology and medicine,
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Metabolism: clinical and experimental,
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Bulletin of experimental biology and medicine,
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Free radical biology & medicine,
