BIOMAT Database Logo BIOMAT Database Logo

Rheumatoid arthritis exacerbation by G-CSF treatment for bucillamine-induced agranulocytosis. 1995

H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho

UI MeSH Term Description Entries
D003545 Cysteine A thiol-containing non-essential amino acid that is oxidized to form CYSTINE. Cysteine Hydrochloride,Half-Cystine,L-Cysteine,Zinc Cysteinate,Half Cystine,L Cysteine
D005260 Female Females
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D000328 Adult A person having attained full growth or maturity. Adults are of 19 through 44 years of age. For a person between 19 and 24 years of age, YOUNG ADULT is available. Adults
D000380 Agranulocytosis A decrease in the number of GRANULOCYTES; (BASOPHILS; EOSINOPHILS; and NEUTROPHILS). Granulocytopenia,Agranulocytoses,Granulocytopenias
D000894 Anti-Inflammatory Agents, Non-Steroidal Anti-inflammatory agents that are non-steroidal in nature. In addition to anti-inflammatory actions, they have analgesic, antipyretic, and platelet-inhibitory actions. They act by blocking the synthesis of prostaglandins by inhibiting cyclooxygenase, which converts arachidonic acid to cyclic endoperoxides, precursors of prostaglandins. Inhibition of prostaglandin synthesis accounts for their analgesic, antipyretic, and platelet-inhibitory actions; other mechanisms may contribute to their anti-inflammatory effects. Analgesics, Anti-Inflammatory,Aspirin-Like Agent,Aspirin-Like Agents,NSAID,Non-Steroidal Anti-Inflammatory Agent,Non-Steroidal Anti-Inflammatory Agents,Nonsteroidal Anti-Inflammatory Agent,Anti Inflammatory Agents, Nonsteroidal,Antiinflammatory Agents, Non Steroidal,Antiinflammatory Agents, Nonsteroidal,NSAIDs,Nonsteroidal Anti-Inflammatory Agents,Agent, Aspirin-Like,Agent, Non-Steroidal Anti-Inflammatory,Agent, Nonsteroidal Anti-Inflammatory,Anti-Inflammatory Agent, Non-Steroidal,Anti-Inflammatory Agent, Nonsteroidal,Anti-Inflammatory Analgesics,Aspirin Like Agent,Aspirin Like Agents,Non Steroidal Anti Inflammatory Agent,Non Steroidal Anti Inflammatory Agents,Nonsteroidal Anti Inflammatory Agent,Nonsteroidal Anti Inflammatory Agents,Nonsteroidal Antiinflammatory Agents
D001172 Arthritis, Rheumatoid A chronic systemic disease, primarily of the joints, marked by inflammatory changes in the synovial membranes and articular structures, widespread fibrinoid degeneration of the collagen fibers in mesenchymal tissues, and by atrophy and rarefaction of bony structures. Etiology is unknown, but autoimmune mechanisms have been implicated. Rheumatoid Arthritis
D016179 Granulocyte Colony-Stimulating Factor A glycoprotein of MW 25 kDa containing internal disulfide bonds. It induces the survival, proliferation, and differentiation of neutrophilic granulocyte precursor cells and functionally activates mature blood neutrophils. Among the family of colony-stimulating factors, G-CSF is the most potent inducer of terminal differentiation to granulocytes and macrophages of leukemic myeloid cell lines. Colony-Stimulating Factor, Granulocyte,G-CSF,Myeloid Growth Factor,Colony Stimulating Factor, Granulocyte,Factor, Granulocyte Colony-Stimulating,Factor, Myeloid Growth,Granulocyte Colony Stimulating Factor,Growth Factor, Myeloid

Related Publications

H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
G-CSF and exacerbation of rheumatoid arthritis.
June 1996, The American journal of medicine,
H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
[A case of rheumatoid arthritis associated with agranulocytosis during bucillamine treatment].
June 1994, Ryumachi. [Rheumatism],
H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
Hyperleucocytosis following G-CSF treatment for sulfasalazine-induced agranulocytosis.
July 2015, BMJ case reports,
H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
Clomipramine-induced agranulocytosis and its treatment with G-CSF.
March 1993, The American journal of psychiatry,
H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
Successful treatment of juvenile rheumatoid arthritis associated haemophagocytic syndrome by cyclosporin A with transient exacerbation by conventional-dose G-CSF.
May 1997, British journal of haematology,
H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
Benefit of G-CSF for methotrexate-induced neutropenia in rheumatoid arthritis.
March 1992, The American journal of medicine,
H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
Treatment of drug-induced agranulocytosis with colony stimulating factors (G-CSF or GM-CSF).
January 1996, Archivum immunologiae et therapiae experimentalis,
H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
Clozapine-induced agranulocytosis and use of g-csf.
January 1998, Indian journal of psychiatry,
H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
The colony-stimulating factors and collagen-induced arthritis: exacerbation of disease by M-CSF and G-CSF and requirement for endogenous M-CSF.
July 2000, Journal of leukocyte biology,
H Nakashima, and K Kawabe, and T Ohtsuka, and K Hayashida, and T Horiuchi, and K Nagasawa, and Y Niho
Asymptomatic interstitial pneumonitis induced by bucillamine in a patient with rheumatoid arthritis.
January 2005, Modern rheumatology,
Copied contents to your clipboard!