-transparent.png){kind=logo}
We defined large striatocapsular infarcts as subcortical softenings of more than 20 mm in diameter involving the territories of the lateral and medial groups of lenticulostriate arteries. The aim of this study of 56 patients was to compare the clinical features and risk factors of these infarcts with those of cortical and lacunar infarcts. On the whole, our data suggest that both the clinical features and risk factors of large striatocapsular infarcts are similar to those of cortical infarcts, but significantly different from those of lacunar infarcts. The clinical manifestations of large striatocapsular infarcts with a maximum diameter of less than 50 mm may sometimes resemble those of lacunar infarcts because neuropsychological disorders are less frequent; however, our study indicates that, even in these cases, cardioembolic sources and artery-to-artery embolism are significantly more frequent in large striatocapsular than in lacunar infarcts, thus suggesting a different pathogenesis.
A Nicolai, and
L G Lazzarino, and
E Biasutti
September 1992,
Acta neurologica Scandinavica,
A Nicolai, and
L G Lazzarino, and
E Biasutti
July 2006,
Stroke,
A Nicolai, and
L G Lazzarino, and
E Biasutti
November 1984,
Neurology,
A Nicolai, and
L G Lazzarino, and
E Biasutti
July 1996,
Journal of neurology,
A Nicolai, and
L G Lazzarino, and
E Biasutti
October 1990,
Archives of neurology,
A Nicolai, and
L G Lazzarino, and
E Biasutti
January 2017,
Frontiers in neurology,
A Nicolai, and
L G Lazzarino, and
E Biasutti
January 2005,
Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association,
A Nicolai, and
L G Lazzarino, and
E Biasutti
October 2009,
Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association,
A Nicolai, and
L G Lazzarino, and
E Biasutti
January 2000,
Cerebrovascular diseases (Basel, Switzerland),
A Nicolai, and
L G Lazzarino, and
E Biasutti
March 2021,
Stroke,
