BIOMAT Database Logo BIOMAT Database Logo

Typical xeroderma pigmentosum complementation group A fibroblasts have detectable ultraviolet light-induced unscheduled DNA synthesis. 1977

R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins

UI MeSH Term Description Entries
D008297 Male Males
D002460 Cell Line Established cell cultures that have the potential to propagate indefinitely. Cell Lines,Line, Cell,Lines, Cell
D002675 Child, Preschool A child between the ages of 2 and 5. Children, Preschool,Preschool Child,Preschool Children
D004247 DNA A deoxyribonucleotide polymer that is the primary genetic material of all cells. Eukaryotic and prokaryotic organisms normally contain DNA in a double-stranded state, yet several important biological processes transiently involve single-stranded regions. DNA, which consists of a polysugar-phosphate backbone possessing projections of purines (adenine and guanine) and pyrimidines (thymine and cytosine), forms a double helix that is held together by hydrogen bonds between these purines and pyrimidines (adenine to thymine and guanine to cytosine). DNA, Double-Stranded,Deoxyribonucleic Acid,ds-DNA,DNA, Double Stranded,Double-Stranded DNA,ds DNA
D004261 DNA Replication The process by which a DNA molecule is duplicated. Autonomous Replication,Replication, Autonomous,Autonomous Replications,DNA Replications,Replication, DNA,Replications, Autonomous,Replications, DNA
D005347 Fibroblasts Connective tissue cells which secrete an extracellular matrix rich in collagen and other macromolecules. Fibroblast
D006801 Humans Members of the species Homo sapiens. Homo sapiens,Man (Taxonomy),Human,Man, Modern,Modern Man
D014466 Ultraviolet Rays That portion of the electromagnetic spectrum immediately below the visible range and extending into the x-ray frequencies. The longer wavelengths (near-UV or biotic or vital rays) are necessary for the endogenous synthesis of vitamin D and are also called antirachitic rays; the shorter, ionizing wavelengths (far-UV or abiotic or extravital rays) are viricidal, bactericidal, mutagenic, and carcinogenic and are used as disinfectants. Actinic Rays,Black Light, Ultraviolet,UV Light,UV Radiation,Ultra-Violet Rays,Ultraviolet Light,Ultraviolet Radiation,Actinic Ray,Light, UV,Light, Ultraviolet,Radiation, UV,Radiation, Ultraviolet,Ray, Actinic,Ray, Ultra-Violet,Ray, Ultraviolet,Ultra Violet Rays,Ultra-Violet Ray,Ultraviolet Black Light,Ultraviolet Black Lights,Ultraviolet Radiations,Ultraviolet Ray
D014983 Xeroderma Pigmentosum A rare, pigmentary, and atrophic autosomal recessive disease. It is manifested as an extreme photosensitivity to ULTRAVIOLET RAYS as the result of a deficiency in the enzyme that permits excisional repair of ultraviolet-damaged DNA. Kaposi Disease,Kaposi's Disease,Kaposis Disease

Related Publications

R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
No lack of complementation for unscheduled DNA synthesis between xeroderma pigmentosum complementation groups D and H.
December 1989, Human genetics,
R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
Xeroderma pigmentosum variants have a slow recovery of DNA synthesis after irradiation with ultraviolet light.
August 1979, Biochimica et biophysica acta,
R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
A xeroderma pigmentosum patient having a high residual activity of unscheduled DNA synthesis after UV is assigned to complementation group A.
November 1976, Mutation research,
R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
Colony-forming ability of ultraviolet-irradiated xeroderma pigmentosum fibroblasts from different DNA repair complementation groups.
August 1976, Biochimica et biophysica acta,
R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
A third complementation group in xeroderma pigmentosum.
January 1974, Mutation research,
R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
A sixth complementation group in xeroderma pigmentosum.
January 1979, Mutation research,
R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
Defective repair of a class of 4NQO-induced alkali-labile DNA lesions in xeroderma pigmentosum complementation group A fibroblasts.
April 1985, Carcinogenesis,
R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
Microinjection of Micrococcus luteus UV-endonuclease restores UV-induced unscheduled DNA synthesis in cells of 9 xeroderma pigmentosum complementation groups.
January 1985, Mutation research,
R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
Xeroderma pigmentosum complementation group H falls into complementation group D.
September 1991, Mutation research,
R A Petinga, and A D Andrews, and R E Tarone, and J H Robbins
Xeroderma pigmentosum complementation group E: a case report.
August 1986, Photo-dermatology,
Copied contents to your clipboard!