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Cimetidine infused intravenously into 6 healthy volunteers did not induce significant changes in plasma levels of prolactin (hPRL), thyreotropin (hTSH), gonadotropin (hLH and hFSH) and growth hormone (hGH). Conversely it caused a significant increase in prolactin response to TRH without modifying the hTSH response to thyreotropin-releasing hormone (TRH) and gonadotropin response to gonadotropin-releasing hormone (GnRH). Data obtained in these experiments, together with data reported in the literature, suggest that the amplified prolactin response to TRH observed during cimetidine infusion, is likely to be connected with a blockade of H2-receptors rather than to unspecific actions of cimetidine.
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
January 1979,
Clinical endocrinology,
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
January 1989,
Sheng li ke xue jin zhan [Progress in physiology],
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
August 1981,
Clinical endocrinology,
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
April 1961,
Deutsche medizinische Wochenschrift (1946),
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
September 1982,
Clinical endocrinology,
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
May 1978,
The Journal of clinical endocrinology and metabolism,
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
January 1986,
Hormone research,
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
January 1955,
Le Progres medical,
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
December 1989,
The Journal of clinical endocrinology and metabolism,
C Scarpignato, and
G Valenti, and
G P Ceda, and
G Bertaccini
November 1978,
Lancet (London, England),
