-transparent.png){kind=logo}
(U)Prostaglandins E-1, E2,F-1alpha or F-2 alpha significantly increased the release of GH, with a parallel increase in intracellular cAMP concentrations, while they only protentiated HE-stimulated TSH release. (2) None of the prostaglandins examined consistently effected either the basal or HE-altered release of LH,FSH or prolactin. (3) The prostaglandin synthetase inhibitor, indomethacin, inhibited GH and TSH release and, at high doses of the drug, inhibited prolactin release. In contrast, the drug appeared to potentiate both He and sLRF-stimulated gonadotropin release. It had no significant effect on intracellular cAMP concentration.
Related Publications
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
January 1976,
Advances in prostaglandin and thromboxane research,
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
January 1974,
Prostaglandins,
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
January 1974,
Prostaglandins,
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
April 1975,
Canadian journal of biochemistry,
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
January 1983,
Fertility and sterility,
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
July 1977,
Nihon Naibunpi Gakkai zasshi,
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
August 1976,
Experientia,
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
November 1991,
The Journal of endocrinology,
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
February 1976,
Prostaglandins,
D K Sundberg, and
C P Fawcett, and
P Illner, and
S M McCann
January 1972,
Advances in cyclic nucleotide research,
