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The most important causes of peptic ulcers are increased gastric acid secretion, impaired mucosal circulation and damages to the mucosa. In contrast, motility plays only a minor role. The following motility disturbances are discussed in peptic ulcers: 1. Impaired gastric emptying may favor the development of gastric ulceration, if it is grossly delayed, and that of duodenal ulceration, if it is inappropriately accelerated. 2. Disturbances of duodenal motility in the fasted state may disturb bicarbonate secretion and, thus, become theoretically ulcerogenic. 3. Duodenogastric reflux has been discussed as a cause of gastric ulceration for a long time; yet, the proof is still missing. 4. According to own studies gastric ulcer is frequently accompanied by morphological and functional changes in the antrum; the cause-effect relationship, however, has yet to be elucidated.
M Wienbeck, and
H J Lübke
July 1992,
Schweizerische Rundschau fur Medizin Praxis = Revue suisse de medecine Praxis,
M Wienbeck, and
H J Lübke
August 1985,
The American journal of medicine,
M Wienbeck, and
H J Lübke
November 1953,
Gazette medicale de France,
M Wienbeck, and
H J Lübke
January 1978,
Minerva chirurgica,
M Wienbeck, and
H J Lübke
January 1991,
Terapevticheskii arkhiv,
M Wienbeck, and
H J Lübke
June 1977,
Klinicheskaia meditsina,
M Wienbeck, and
H J Lübke
January 1992,
Terapevticheskii arkhiv,
M Wienbeck, and
H J Lübke
January 1976,
Khirurgiia,
M Wienbeck, and
H J Lübke
January 1991,
Pediatriia,
M Wienbeck, and
H J Lübke
November 1963,
Sovetskaia meditsina,
