Thromboxane synthetase inhibition and thromboxane receptor blockade preserve pulmonary and circulatory function in a porcine burn sepsis model. 1994

G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
Shriners Burns Institute, Galveston, TX.

BACKGROUND Thromboxane A2 (TxA2) is a key mediator in the pathophysiology of severe burns and sepsis. This study was performed to assess the potential benefits of TxA2 synthetase inhibition and TxA2 receptor blockade in sepsis after severe thermal injury. METHODS Pigs with left atrial, aortic, and pulmonary artery catheters received a 40 percent third-degree burn and, 24 hours later, 100 micrograms per kg Escherichia coli endotoxin. The antagonist treatment (BM) group was treated with the TxA2 receptor antagonist BM 13.177, the synthetase treatment (OKY) group with the TxA2 synthetase inhibitor OKY-046, and the control group received saline solution placebo. Another group without burn or endotoxin was used to assess the side effects of BM 13.177. RESULTS Both drugs significantly attenuated the changes in pulmonary vascular resistance index, cardiac index, arterial PO2, shunt, oxygen delivery, and oxygen consumption seen after endotoxin. However, cardiac index was significantly decreased in group BM before endotoxin. In healthy pigs, BM 13.177 decreased cardiac index and oxygen delivery and increased the pulmonary vascular resistance index. CONCLUSIONS TxA2 synthetase inhibitors and TxA2 receptor blockers are potentially useful in sepsis after severe burns. Comparison between drugs was complicated by the adverse effects of the antagonist, and further investigation with other antagonists is needed.

UI MeSH Term Description Entries
D008070 Lipopolysaccharides Lipid-containing polysaccharides which are endotoxins and important group-specific antigens. They are often derived from the cell wall of gram-negative bacteria and induce immunoglobulin secretion. The lipopolysaccharide molecule consists of three parts: LIPID A, core polysaccharide, and O-specific chains (O ANTIGENS). When derived from Escherichia coli, lipopolysaccharides serve as polyclonal B-cell mitogens commonly used in laboratory immunology. (From Dorland, 28th ed) Lipopolysaccharide,Lipoglycans
D008168 Lung Either of the pair of organs occupying the cavity of the thorax that effect the aeration of the blood. Lungs
D008689 Methacrylates Acrylic acids or acrylates which are substituted in the C-2 position with a methyl group. Methacrylate
D010101 Oxygen Consumption The rate at which oxygen is used by a tissue; microliters of oxygen STPD used per milligram of tissue per hour; the rate at which oxygen enters the blood from alveolar gas, equal in the steady state to the consumption of oxygen by tissue metabolism throughout the body. (Stedman, 25th ed, p346) Consumption, Oxygen,Consumptions, Oxygen,Oxygen Consumptions
D011651 Pulmonary Artery The short wide vessel arising from the conus arteriosus of the right ventricle and conveying unaerated blood to the lungs. Arteries, Pulmonary,Artery, Pulmonary,Pulmonary Arteries
D011659 Pulmonary Gas Exchange The exchange of OXYGEN and CARBON DIOXIDE between alveolar air and pulmonary capillary blood that occurs across the BLOOD-AIR BARRIER. Exchange, Pulmonary Gas,Gas Exchange, Pulmonary
D001775 Blood Circulation The movement of the BLOOD as it is pumped through the CARDIOVASCULAR SYSTEM. Blood Flow,Circulation, Blood,Blood Flows,Flow, Blood
D001794 Blood Pressure PRESSURE of the BLOOD on the ARTERIES and other BLOOD VESSELS. Systolic Pressure,Diastolic Pressure,Pulse Pressure,Pressure, Blood,Pressure, Diastolic,Pressure, Pulse,Pressure, Systolic,Pressures, Systolic
D002056 Burns Injuries to tissues caused by contact with heat, steam, chemicals (BURNS, CHEMICAL), electricity (BURNS, ELECTRIC), or the like. Burn
D002302 Cardiac Output The volume of BLOOD passing through the HEART per unit of time. It is usually expressed as liters (volume) per minute so as not to be confused with STROKE VOLUME (volume per beat). Cardiac Outputs,Output, Cardiac,Outputs, Cardiac

Related Publications

G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
August 2012, Acta physiologica (Oxford, England),
G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
February 1989, American journal of kidney diseases : the official journal of the National Kidney Foundation,
G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
March 1987, Chest,
G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
January 1989, Progress in clinical and biological research,
G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
May 1989, Surgery,
G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
January 1988, Acta physiologica Hungarica,
G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
April 1996, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation,
G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
June 1989, Archives of surgery (Chicago, Ill. : 1960),
G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
December 2002, Pediatric research,
G Iglesias, and S T Zeigler, and C W Lentz, and D L Traber, and D N Herndon
December 1984, Thrombosis and haemostasis,
Copied contents to your clipboard!